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Squalene acts as a feedback signaling molecule in facilitating bidirectional communication between tea plants. Sci Adv. 2025. 11(7): eads4888.
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Evolution and functional divergence of glycosyltransferase genes shaped the quality and cold tolerance of tea plants. Plant Cell. 2024. 37(1): koae268.
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2,4-Dihydroxybenzoic Acid, a Novel SA Derivative, Controls Plant Immunity via UGT95B17-Mediated Glucosylation: A Case Study in Camellia Sinensis. Adv Sci (Weinh). 2024. 11(7): e2307051.
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Drought-Triggered JA Signaling Promotes Pathogen Susceptibility by Attenuating Lignin Defenses via UGT72BV9 in Tea Plants. Plant Physiol. 2026. 201(4): kiag559.
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Precise control of volatile glucosylation in tea plants by CBF4, WRKY4, and an atypical bHLH transcription factor. Plant Physiol. 2026. 201(3): kiag145.
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Cold stress-induced (Z)-3-hexenol and thymol enhance cold tolerance of tea plants by activating Ca2+ signaling. Plant Biotechnology Journal. 2025. 23(12): 5833-5848.
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A calcium-decoding module translates volatile DMNT into jasmonic acid-mediated herbivore resistance in Camellia sinensis. Plant Physiology. 2025. 199(1): kiaf386.
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Tea aphid-induced β-Elemene biosynthesis by CsELE enhances JA-dependent herbivore resistance in tea plants. Plant Cell Environ. 2025. 48(9): 6473-6489.
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Natural variation of CsUGT71A60 determines growth and cold tolerance via regulating cytokinin glycosylation in Camellia sinensis. Plant Biotechnol J. 2025. 23(7): 2809-2823.
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Glycoside-specific metabolomics reveals the novel mechanism of glycinebetaine-induced cold tolerance by regulating apigenin glycosylation in tea plants. New Phytologist. 2025. 245(6): 2616-2631.
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UGT89AC1‐mediated quercetin glucosylation is induced upon herbivore damage and enhances Camellia sinensis resistance to insect feeding. Plant Cell Environ. 2024. 47(2): 682-697.
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(Z)-3-hexenol integrates drought and cold stress signaling by activating abscisic acid glucosylation in tea plants. Plant Physiol. 2023. 193(2): 1491-1507.
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育种技术与品种培育
茶树基因组
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